NEPHROTIC SYNDROME

Nephrotic Syndrome is not a specific kidney disorder. It is not a disease itself but rather refers to a group of symptoms including Edema( around the eyes, feet and hands), Proteinuria (protein in urine), Hyperlipidaemia (high lipid levels),and Hypoalbuminemia ( is a medical condition where blood levels of albumin are abnormally low). It more commonly occurs in children than adults. It can have hereditary factors also.As the filtering units of the kidneys(nephrons) get inflammed and impaired in this disorder; this damage allows protein normally which is kept in the plasma to leak into the urine in large amounts (proteinuria at least 3.5 grams per day per 1.73m2 body surface area) , thus resulting in Nephrotic Syndrome. Since the protein in the blood helps keep fluid in the bloodstream, some of this fluid leaks out of the bloodstream into cells of the tissues in patient’s body, causing swelling, called edema.

It can occur with many diseases, so prevention relies on controlling the diseases that cause it. Treatment of the nephrotic syndrome focuses on identifying and treating the underlying cause, if possible, and reducing high cholesterol, blood pressure, and protein in the urine through diet, medication, or both. The nephrotic syndrome may go away once the underlying cause, if known, is treated. However, often a kidney disease is the underlying cause and in allopathy, it cannot be cured. In these cases, the kidneys may gradually lose their ability to filter wastes and excess water from the blood. If kidney failure occurs, the patient will need to be on dialysis or have a kidney transplant.

OTHER CHARACTERISTICS OF NEPHROTIC SYNDROME:

• Puffiness around the eyes, characteristically in the morning.
• Pitting edema over the legs.
• Fluid in the pleural cavity causing pleural effusion. More commonly associated with excess fluid is pulmonary edema.
• Fluid in the peritoneal cavity causing ascites.
• Generalized edema throughout the body known as anasarca.
• Most of the patients are normotensive but hypertension (rarely) may also occur.
• Anemia (iron resistant microcytic hypochromic type) maybe present due to transferrin loss.
• Dyspnea (Shortness of breath) maybe present due to pleural effusion or due to diaphragmatic compression with ascites.
• Erythrocyte sedimentation rate (ESR) is increased due to increased fibrinogen & other plasma contents.
• Some patients may notice foamy or frothy urine, due to a lowering of the surface tension by the severe proteinuria. Actual urinary complaints such as hematuria or oliguria are uncommon, though these are seen commonly in nephritic syndrome.
• May have features of the underlying cause, such as the rash associated with systemic lupus erythematosus, or the neuropathy associated with diabetes.

AYURVEDIC TREATMENT

In Allopathy, proper cause as well as cure of the disease is not known and the line of treatment is the administration of various kinds of corticosteroids which temporarily decrease the excretion of protein in the urine and hold for a while, further complications associated with it. Immunosuppressive drugs are given to stop proteinuria in some patients, but once the treatments have ended proteinuria will continue. The drugs may sometimes damage the patient’s kidneys even more.

Patients can have a remission after a period of steroid therapy. However, frequent relapse is a typical sign of Nephrotic Syndrome. So, there is no long term sustainable results and cure of Nephrotic Syndrome available in Allopathy.

Whereas in Ayurveda, we dig at the cause of the disease to treat the underlying cause and not just try to suppress the symptoms. Ayurveda believes that this condition is caused due to the vitiation of Pitta dosha in the body and various Pitta normalizing medications are used in the treatment of the same

Ayurvedic treatment is a traditional treatment in India and works on the principle of targeting the cause and not symptoms, taking into consideration the prakriti and dosha equilibrium of the patient.

Some of these medications include:

• Chandanasavam
• Chandaanaadi Wati

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